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Carcinogenesis Advance Access originally published online on September 16, 2009
Carcinogenesis 2009 30(11):1910-1915; doi:10.1093/carcin/bgp224
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© The Author 2009. Published by Oxford University Press. All rights reserved. For Permissions, please email: journals.permissions@oxfordjournals.org

A polymorphic variant in human MDM4 associates with accelerated age of onset of estrogen receptor negative breast cancer

Diptee A. Kulkarni, Alexei Vazquez1,2, Bruce G. Haffty2, Elisa V. Bandera3, Wenwei Hu4, Yvonne Y. Sun4, Deborah L. Toppmeyer, Arnold J. Levine1,4 and Kim M. Hirshfield*

Department of Medicine-Division of Medical Oncology, The Cancer Institute of New Jersey, University of Medicine and Dentistry of New Jersey-Robert Wood Johnson Medical School, New Brunswick, NJ 08901, USA
1 The Simons Center for Systems Biology, Institute for Advanced Study, Princeton, NJ 08540, USA
2 Department of Radiation Oncology
3 Department of Surgical Oncology
4 Department of Pediatrics, The Cancer Institute of New Jersey, University of Medicine and Dentistry of New Jerse-Robert Wood Johnson Medical School, New Brunswick, NJ 08901, USA

* To whom correspondence should be addressed. Tel: +1 732 235 6028; Fax: +1 732 235 5331; Email: hirshfie{at}umdnj.edu

Murine double minute 4 (MDM4) shares significant structural homology with murine double minute 2 (MDM2) and interacts and regulates transcriptional activity of the tumor suppressor p53. In tumors with wild-type p53, there is often overexpression of MDM2 or MDM4 leading to functional inactivation of p53. A single-nucleotide polymorphism (SNP) in the promoter of human MDM2 (SNP309) was shown to associate with increased MDM2 expression and increased risk of cancer. This study evaluated the association of a SNP in human MDM4 (C>T) with age of onset of breast cancer in two independent cohorts. In cohort 1 of 675 patients, the average age of diagnosis for women with estrogen receptor (ER)-positive and ER-negative breast cancers was 53.2 and 48 years, respectively. In this cohort, homozygous variant (TT) carriers developed ER-negative carcinomas at an earlier age than homozygous wild-type (CC) or heterozygous (TC) such that the age at diagnosis was accelerated by 5.0 years (P = 0.018). This association was validated in a second cohort of breast cancer patients (n = 148), where TT carriers with ER-negative cancer developed the disease 3.8 years earlier than CC carriers (P = 0.006). The effect was more pronounced in Caucasians with ER-negative ductal carcinomas with TT homozygotes developing disease 7.5 years (P = 0.031) and 6.2 years (P = 7 x 10–5) earlier than CC carriers in cohorts 1 and 2, respectively. No association was seen in ER-positive ductal cancers suggesting that the SNP in MDM4 only has a functional association in ER-negative breast cancer.

Abbreviations: ER, estrogen receptor; MDM2, murine double minute 2; MDM4, murine double minute 4; SNP, single-nucleotide polymorphism

Received June 18, 2009; revised August 19, 2009; accepted September 5, 2009.


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