Carcinogenesis, Vol. 24, No. 1, 147,
January 2003
© 2003 Oxford University Press
RESPONSE TO LETTER TO THE EDITOR |
Cag A status of Helicobacter pylori infection and p53 gene mutations in gastric adenocarcinoma
1 Department of Health Research and Policy,
2 Department of Medicine and
3 Department of Pathology, Stanford University School of Medicine, Stanford, CA 94305, USA,
4 Department of Pathology and
5 Division of Research, Kaiser Permanente Medical Care Program, Oakland, CA 94611, USA,
6 Metabolism Branch, National Cancer Institute, Bethesda, MD 20892, USA and
7 Orentreich Foundation for the Advancement of Science, Inc., Cold Spring-on-Hudson, NY 10516, USA
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Dear Sir,
We thank Magalhães et al. for their interest in our study and for reminding us of the complexity inherent in the interpretation of our results (1). We agree that a single measurement