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Carcinogenesis Advance Access published online on February 17, 2008

Carcinogenesis, doi:10.1093/carcin/bgn051
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© The Author 2008. Published by Oxford University Press. All rights reserved. For Permissions, please email: journals.permissions@oxfordjournals.org

Protective versus promotional effects of white tea and caffeine on PhIP-induced tumorigenesis and β-catenin expression in the rat

Rong Wang1, W. Mohaiza Dashwood1, Christiane V. Lohr2, Kay A. Fischer2, Clifford B. Pereira3, Mandy Louderback1, Hitoshi Nakagama4, George S. Bailey1, David E. Williams1,5 and Roderick H. Dashwood1,5,*

1 Linus Pauling Institute, Oregon State University, Corvallis OR 97331 USA
2 College of Veterinary Medicine, Oregon State University, Corvallis OR 97331 USA
3 Department of Statistics, Oregon State University, Corvallis OR 97331 USA
4 Biochemistry Division, National Cancer Center Research Institute, 1-1, Tsukiji 5-chome, Chuo-ku, Tokyo 104-0045, Japan
5 Department of Environmental and Molecular Toxicology, Oregon State University, Corvallis OR 97331, USA

* To whom correspondence should be addressed at Linus Pauling Institute, Oregon State University, Corvallis, OR 97331-6512, USA. Tel: +541 737 5086; Fax: +541 737 5077; Email Rod.Dashwood{at}oregonstate.edu

A one-year carcinogenicity bioassay was conducted in rats treated with three short cycles of 2-amino-1-methyl-6-phenylimidazo[4,5-b]pyridine (PhIP)/high-fat diet (HF), followed by 2% white tea (w/v), 0.05% epigallocatechin-3-gallate (EGCG), or 0.065% caffeine as sole source of fluid intake. Thirty-two percent of the PhIP/HF controls survived to 1 year, compared with 50%, 48.7%, and 18.2% in groups given white tea, EGCG, and caffeine, respectively. After 1 year, PhIP/HF controls had tumors in the colon, skin, small intestine, Zymbal's gland, salivary gland, and pancreas. For all sites combined, excluding the colon, tumor incidence data were as follows: PhIP/HF 69.5%, PhIP/HF+EGCG 48.7%, PhIP/HF+white tea 46.9%, and PhIP/HF+caffeine 13.3%. Unexpectedly, a higher incidence of colon tumors was detected in rats post-treated with white tea (69%) and caffeine (73%), compared with the 42% incidence in PhIP/HF controls. In the colon tumors, β-catenin mutations were detected at a higher frequency after caffeine post-treatment, and there was a shift towards more tumors harboring substitutions of Gly34, with correspondingly high protein and mRNA expression seen for both β-catenin and c-Myc. c-Myc expression exhibited concordance with tumor promotion, and there was a concomitant increase in cell proliferation versus apoptosis in colonic crypts. A prior report described suppression of PhIP-induced colonic aberrant crypts by the same test agents, but did not incorporate a high-fat diet. These findings are discussed in the context of epidemiological data which do not support an adverse effect of tea and coffee on colon tumor outcome – indeed, some such studies suggest a protective role for caffeinated beverages

Received January 16, 2008; revised February 4, 2008; accepted February 8, 2008.


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